Vets highlight water toxicities
Providing access to water can be challenging, especially for operations enduring drought conditions. Water-related toxicities and fatalities may be seen with greater frequency under hot and dry time periods.
Warnings of toxicity often develop rapidly, and animal death may be the first clinical sign. Common water-related toxicities include water deprivation, sulfates, blue-green algae and nitrates.
The common history of water deprivation is typically when an automatic water stops functioning, or the pump at the well quits working unbeknownst to the rancher. With good intentions, the cattle are given immediate and full access to water.
Unfortunately, this approach often leads to rapid imbalances in electrolytes and salt toxicosis. It is important to note, salt toxicosis in this instance is not the over consumption of salt.
Animals having been without water have elevated sodium levels in their tissues, including the brain. When animals are given free or rapid access to water, the fluid rushes to the high levels of sodium and essentially floods the brain. Cattle rapidly show neurologic signs including incoordination, weakness, seizures and death.
If animals have been deprived, rewatering should be done gradually over time. Just a few inches of water should be placed in a tank or trough. Depending on the conditions, rewatering may need to be done over a period of several hours. Consultation with a veterinarian is the best approach.
Cattle experiencing sulfur toxicity also show neurological signs. Commonly, veterinarians would call this condition polioencephalomalacia or polio.
Cattle initially become depressed, decrease feed intake and exhibit lethargy. As the condition progresses, they become ataxic, stagger and may die.
It is advisable to test water sources for sulfate levels and retesting may need to be done over time as levels can change. Additionally, sulfate levels are additive, so both feed and water should be assessed to determine acceptable levels.
The classic history of blue-green algae (BGA), also known as cyanobacteria, is several dead animals surrounding a pond. Not all species of BGA are toxic.
BGA produce a toxin leading to neurologic signs and rapid death usually within a few hours. Liver damage which may be noticed as photosensitivity or bloody diarrhea can also be seen.
In these cases, water should always be tested for a BGA bloom. Fertilizer and manure runoff can increase chances of BGA.
Ponds should be monitored closely for signs of bloom and dead wildlife. If BGA is suspected, all remaining animals in the pasture should be moved to a location with a different water source.
Nitrate toxicity is often part of the discussion when evaluating forages, especially Johnson grass. However, a common history for nitrate toxicity includes hauling of water in containers previously used for fertilizer.
Due to the concentration of fertilizers or other chemicals, it is nearly impossible to remove residue to a safe level. Nitrates may also reach dangerous levels in ponds or other water sources which collect drainage from manure, highly fertilized fields or industrial waste.
Neurologic signs and death are also seen with nitrates as have been previously described with other toxicities. The blood in nitrate toxicity is chocolate brown.
Just as with sulfates, the effects of nitrate levels of forage, feed and water are additive. So, both feed and water must be considered when evaluating a nitrate problem.
Deep wells are usually safe sources of water. No safety standards have been set for livestock water, but it has been suggested up to 100 parts per million of nitrate nitrogen in water should be safe if cattle are consuming an adequate ration free of nitrates. This is 10 times the safe level set for humans.
Oklahoma State University has laboratories which can assist producers in water testing. Information on the Oklahoma Animal Disease Diagnostic Laboratory can be found at vetmed.okstate.edu/oaddl. Information on the Soil, Water and Forage Analytic Laboratory can be found at soiltesting.okstate.edu.
Information in this article was previously featured June 29 on feedlotmagazine.com. It is courtesy of Oklahoma State University’s College of Veterinary Medicine Beef Extension Specialist Rosslyn Biggs, DVM and Assistant Professor in the Department of Veterinary Pathobiology Brianne Taylor, DVM. Send comments on this article to email@example.com.